| Categories genes  | Tags pollen  anther  development  cell death  fertility  tapetal  anther development  PCD  reactive oxygen species  tapetal programmed cell death 
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  • Key message
    • Knockdown of OsAGO2 led to the overaccumulation of reactive oxygen species (ROS) and abnormal anther development, causing premature initiation of tapetal programmed cell death (PCD) and pollen abortion
    • Moreover, knockdown of OsHXK1 restored pollen fertility in OsAGO2 knockdown plants
    • Here, we show that the AGO protein OsAGO2 regulates anther development in rice
    • OsAGO2 controls ROS production and the initiation of tapetal PCD by epigenetically regulating OsHXK1 expression in rice anthers.
    • These results indicate that OsHXK1 controls the appropriate production of ROS and the proper timing of tapetal PCD and is directly regulated by OsAGO2 through epigenetic regulation
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