- Information
- Symbol: OsbHLH98
- MSU: LOC_Os03g58330
- RAPdb: Os03g0797600
- Publication
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Genbank accession number
- Key message
- OsbHLH98 regulates leaf angle in rice through transcriptional repression of OsBUL1.
- osbhlh98 mutant leaves formed a larger leaf angle, whereas transgenic plants overexpressing OsbHLH98 exhibited a slight reduction in leaf angle
- We determined that the changes in leaf angle resulted from increased number and size of parenchyma cells on the adaxial side of the lamina joint in osbhlh98 mutants
- Experiments using reporter constructs showed that OsbHLH98 is expressed on the adaxial side of lamina joints, consistent with its proposed function in regulating leaf angle
- Our results demonstrate that OsbHLH98 negatively regulates leaf angle by counteracting brassinosteroid-induced cell elongation via the repression of OsBUL1 transcription
- The characterization of OsbHLH98 and its role in determining leaf angle will lay the foundation to develop the ideal plant architecture for adaptation to high planting density
- Connection
- OsbHLH98, PGL2~OsBUL1~OsbHLH173, OsbHLH98 regulates leaf angle in rice through transcriptional repression of OsBUL1., OsbHLH98 regulates leaf angle in rice through transcriptional repression of OsBUL1.
- OsbHLH98, PGL2~OsBUL1~OsbHLH173, OsbHLH98 regulates leaf angle in rice through transcriptional repression of OsBUL1., Furthermore, we established by chromatin immunoprecipitation and CUT&RUN that OsBUL1 is a direct downstream target of OsbHLH98
- OsbHLH98, PGL2~OsBUL1~OsbHLH173, OsbHLH98 regulates leaf angle in rice through transcriptional repression of OsBUL1., Transactivation assays and reverse transcription quantitative polymerase chain reaction (RT-qPCR) analysis indicated that OsbHLH98 represses OsBUL1 transcription
- OsbHLH98, PGL2~OsBUL1~OsbHLH173, OsbHLH98 regulates leaf angle in rice through transcriptional repression of OsBUL1., Our results demonstrate that OsbHLH98 negatively regulates leaf angle by counteracting brassinosteroid-induced cell elongation via the repression of OsBUL1 transcription
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