- Information
- Symbol: OsRR1
- MSU: LOC_Os04g36070
- RAPdb: Os04g0442300
- Publication
- Identification and characterization of cytokinin-signalling gene families in rice, 2006, Gene.
- Molecular characterization and differential expression of cytokinin-responsive type-A response regulators in rice Oryza sativa, 2006, BMC Plant Biol.
- The auxin responsive AP2/ERF transcription factor CROWN ROOTLESS5 is involved in crown root initiation in rice through the induction of OsRR1, a type-A response regulator of cytokinin signaling, 2011, Plant J.
- Homodimerization of Ehd1 is required to induce flowering in rice., 2016, Plant Physiol.
- Transcriptional and post-transcriptional regulation of heading date in rice, 2020, New Phytol.
- Endoplasmic Reticulum-Localized PURINE PERMEASE1 Regulates Plant Height and Grain Weight by Modulating Cytokinin Distribution in Rice, 2020, Front Plant Sci.
- Genbank accession number
- Key message
- Taken together, these results indicate that auxin-induced CRL5 promotes crown root initiation through repression of cytokinin signaling by positively regulating type-A RR, OsRR1
- The auxin responsive AP2/ERF transcription factor CROWN ROOTLESS5 is involved in crown root initiation in rice through the induction of OsRR1, a type-A response regulator of cytokinin signaling
- In addition, ProACT:CRL5/WT showed a cytokinin-resistant phenotype for crown root initiation, and also up-regulated the expression of two negative regulators of cytokinin signaling, OsRR1 and OsRR2, which were downregulated in crl5
- Transgenic plants that over-expressed OsRR1 under the control of the CRL5 promoter in a crl5 mutant background produced a higher number of crown roots than the crl5 plant
- sativa RR) showed cytokinin-induced expression, and three (OsRR8, OsRR12 and OsRR13) showed expression in flower
- Connection
- Crl5, OsRR1, The auxin responsive AP2/ERF transcription factor CROWN ROOTLESS5 is involved in crown root initiation in rice through the induction of OsRR1, a type-A response regulator of cytokinin signaling, In addition, ProACT:CRL5/WT showed a cytokinin-resistant phenotype for crown root initiation, and also up-regulated the expression of two negative regulators of cytokinin signaling, OsRR1 and OsRR2, which were downregulated in crl5
- Crl5, OsRR1, The auxin responsive AP2/ERF transcription factor CROWN ROOTLESS5 is involved in crown root initiation in rice through the induction of OsRR1, a type-A response regulator of cytokinin signaling, Transgenic plants that over-expressed OsRR1 under the control of the CRL5 promoter in a crl5 mutant background produced a higher number of crown roots than the crl5 plant
- Crl5, OsRR1, The auxin responsive AP2/ERF transcription factor CROWN ROOTLESS5 is involved in crown root initiation in rice through the induction of OsRR1, a type-A response regulator of cytokinin signaling, Taken together, these results indicate that auxin-induced CRL5 promotes crown root initiation through repression of cytokinin signaling by positively regulating type-A RR, OsRR1
- OsRR1, OsRR2, The auxin responsive AP2/ERF transcription factor CROWN ROOTLESS5 is involved in crown root initiation in rice through the induction of OsRR1, a type-A response regulator of cytokinin signaling, In addition, ProACT:CRL5/WT showed a cytokinin-resistant phenotype for crown root initiation, and also up-regulated the expression of two negative regulators of cytokinin signaling, OsRR1 and OsRR2, which were downregulated in crl5
- Ehd1, OsRR1, Homodimerization of Ehd1 is required to induce flowering in rice., A type-A RR, OsRR1, physically interacts with Ehd1 to form a heterodimer
- Ehd1, OsRR1, Homodimerization of Ehd1 is required to induce flowering in rice., Based on these observations, we conclude that OsRR1 inhibits Ehd1 activity by binding to form an inactive complex
- OsAFB6~OsAFB5, OsRR1, Overexpression of an auxin receptor OsAFB6 significantly enhanced grain yield by increasing cytokinin and decreasing auxin concentrations in rice panicle., OsAFB6 is preferentially expressed in young tissues with stronger meristem activities and suppresses flowering by upregulating OsRR1 and downregulating Ehd1 expression levels
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