- Information
- Symbol: OsGORK,OsK5.2
- MSU: LOC_Os06g14030
- RAPdb: Os06g0250600
- Publication
- Differential gene expression of two outward-rectifying shaker-like potassium channels OsSKOR and OsGORK in rice, 2015, Journal of Plant Biology.
- A dual role for the OsK5.2 ion channel in stomatal movements and K+ loading into xylem sap., 2017, Plant Physiol.
- Tissue-Specific Regulation of Na+ and K+ Transporters Explains Genotypic Differences in Salinity Stress Tolerance in Rice., 2019, Front Plant Sci.
- The outward shaker channel OsK5.2 improves plant salt tolerance by contributing to control of both leaf transpiration and K+ secretion into xylem sap., 2022, Plant Cell Environ.
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Genbank accession number
- Key message
- OsGORK was expressed to some degree in most tissues, such as leaf blade, node, leaf sheath and root, but at high levels in flowers
- Differential gene expression of two outward-rectifying shaker-like potassium channels OsSKOR and OsGORK in rice
- A dual role for the OsK5.2 ion channel in stomatal movements and K+ loading into xylem sap.
- This trait was conferred by at least three complementary mechanisms: (1) its superior ability to activate H+-ATPase pump operation, both at transcriptional and functional levels; (2) reduced sensitivity of K+ efflux channels to reactive oxygen species; and (3) smaller upregulation in OsGORK and higher upregulation of OsAKT1 in tolerant cultivars in response to salt stress
- The outward shaker channel OsK5.2 improves plant salt tolerance by contributing to control of both leaf transpiration and K(+) secretion into xylem sap.
- Connection
- OsGORK~OsK5.2, OsNAC2~OsTIL1~OMTN2~OsORE1, OsNAC2 positively affects salt-induced cell death and binds to the OsAP37 and OsCOX11 promoters., Furthermore, K+-efflux channels (OsGORK and OsSKOR) were obviously activated by OsNAC2
- OsAKT1, OsGORK~OsK5.2, Tissue-Specific Regulation of Na+ and K+ Transporters Explains Genotypic Differences in Salinity Stress Tolerance in Rice., This trait was conferred by at least three complementary mechanisms: (1) its superior ability to activate H+-ATPase pump operation, both at transcriptional and functional levels; (2) reduced sensitivity of K+ efflux channels to reactive oxygen species; and (3) smaller upregulation in OsGORK and higher upregulation of OsAKT1 in tolerant cultivars in response to salt stress
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