- Information
- Symbol: OsMIR168a
- MSU: None
- RAPdb: None
- Publication
- CRISPR-Cas9 mediated OsMIR168a knockout reveals its pleiotropy in rice., 2021, Plant Biotechnol J.
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Genbank accession number
- Key message
- The rice OsMIR168a mutants grew rapidly at the seedling stage, produced more tillers, and matured early
- Many protein-coding genes and miRNAs showed differential expression in the OsMIR168a mutants, suggesting OsMIR168a exerts a major transcriptional regulatory role, likely through its potential target genes such as OsAGO1s and OsAGO18
- Connection
- AGO18~OsAGO18, OsMIR168a, CRISPR-Cas9 mediated OsMIR168a knockout reveals its pleiotropy in rice., Intriguingly, OsAGO18, a member of a new AGO clade that is conserved in monocots, was confirmed to be a target of OsMIR168a not only by informatic prediction but also by expression analysis and a cell-based cleavage assay in the OsMIR168a mutants
- AGO18~OsAGO18, OsMIR168a, CRISPR-Cas9 mediated OsMIR168a knockout reveals its pleiotropy in rice., Many protein-coding genes and miRNAs showed differential expression in the OsMIR168a mutants, suggesting OsMIR168a exerts a major transcriptional regulatory role, likely through its potential target genes such as OsAGO1s and OsAGO18
- OsAGO1a, OsMIR168a, CRISPR-Cas9 mediated OsMIR168a knockout reveals its pleiotropy in rice., Analysis of gene expression showed that the transcription levels of OsMIR168a's target genes such as OsAGO1a, OsAGO1b and OsAGO1d were elevated significantly in the OsMIR168a mutants
- OsAGO1b, OsMIR168a, CRISPR-Cas9 mediated OsMIR168a knockout reveals its pleiotropy in rice., Analysis of gene expression showed that the transcription levels of OsMIR168a's target genes such as OsAGO1a, OsAGO1b and OsAGO1d were elevated significantly in the OsMIR168a mutants
- OsAGO1d, OsMIR168a, CRISPR-Cas9 mediated OsMIR168a knockout reveals its pleiotropy in rice., Analysis of gene expression showed that the transcription levels of OsMIR168a's target genes such as OsAGO1a, OsAGO1b and OsAGO1d were elevated significantly in the OsMIR168a mutants
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