- Information
- Symbol: OsRAV2
- MSU: LOC_Os01g04800
- RAPdb: Os01g0141000
- Publication
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Genbank accession number
- Key message
- Identification of a regulatory element responsible for salt induction of rice OsRAV2 through ex situ and in situ promoter analysis.
- Transgenic analysis indicated that P OsRAV2 is induced by salt stress but not osmotic stress or ABA treatment
- Serial 5’ deletions and site-specific mutations in P OsRAV2 revealed that a GT-1 element located at position -664 relative to the putative translation start site is essential for the salt induction of P OsRAV2
- The regulatory function of the GT-1 element in the salt induction of OsRAV2 was verified in situ in plants with targeted mutations generated using the CRISPR/Cas9 (clustered regularly interspaced short palindromic repeats/CRISPR-associated protein 9) system
- Taken together, our results indicate that the GT-1 element directly controls the salt response of OsRAV2
- Further expression profile analyses indicated that OsRAV2 is transcriptionally regulated by salt, but not KCl, osmotic stress, cold or ABA (abscisic acid) treatment
- To elucidate the regulatory mechanism of the stress response at the transcriptional level, we isolated and characterized the promoter region of OsRAV2 (P OsRAV2 )
- Connection
- OsRAV2, __SAB18~OsGT
-2__, [The trihelix transcription factor OsGTγ-2 is involved adaption to salt stress in rice.](http://www.ncbi.nlm.nih.gov/pubmed?term=The trihelix transcription factor OsGTγ-2 is involved adaption to salt stress in rice.%5BTitle%5D), Here, we identified a rice OsGTγ family member, OsGTγ-2, which directly interacted with the GT-1 element in the OsRAV2 promoter
- OsRAV2, __SAB18~OsGT
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